Neurology · Year 3 · from Neurology

Case 2: Neuromyelitis Optica Spectrum Disorder (NMOSD)

Patient Demographics

  • Age: 35 years
  • Sex: Female
  • Occupation: Social worker
  • Ethnicity: African American

Chief Complaint

"I went blind in both eyes and can't move my legs."

History of Present Illness

The patient presented 10 days ago with severe bilateral vision loss and weakness in both legs that progressed to complete paralysis over 48 hours. She had intractable nausea, vomiting, and hiccups for 2 weeks before the current symptoms (area postrema syndrome). She also developed urinary retention requiring catheterization. Three years ago, she had an episode of bilateral leg weakness and numbness that was diagnosed as "transverse myelitis" and treated with steroids with partial recovery. She has a history of systemic lupus erythematosus (SLE). No similar symptoms in family.

Neurological Examination Findings

Mental Status:

  • Alert, oriented, distressed

Cranial Nerves:

  • CN II (bilateral):
  • Visual acuity: Light perception only in both eyes (severe bilateral visual loss)
  • RAPD: Difficult to assess with bilateral involvement
  • Color vision: Unable to test
  • Fundoscopy: Bilateral disc edema
  • Extraocular movements: Intact
  • Intractable hiccups during examination

Motor Examination:

  • Upper extremities: 5/5 bilateral
  • Lower extremities: 0/5 bilateral (complete paraplegia)
  • Hypotonic legs (acute stage)

Sensory Examination:

  • Complete loss of all sensory modalities below T4 level
  • Sensory level at T4

Reflexes:

  • Upper extremities: 2+
  • Lower extremities: Absent (spinal shock)
  • Babinski: Mute initially (may become positive after spinal shock resolves)

Other:

  • Urinary retention - Foley in place

NMOSD vs MS: Key Differentiating Features

FeatureThis Patient (NMOSD)Typical MS
Optic neuritisBilateral, severeUnilateral, moderate
MyelitisLongitudinally extensive (≥3 segments)Short segment (<2 segments)
RecoveryPoorUsually good
MRI brainNormal or atypical lesionsTypical periventricular lesions
EthnicityMore common in non-white populationsMore common in white populations
Associated autoimmunityCommon (SLE, Sjogren's)Uncommon

Neuro Workup

  • MRI Spine:
  • Longitudinally extensive transverse myelitis (LETM): T2 hyperintensity extending from C2-T6 (>3 vertebral segments)
  • Central cord predominance
  • Enhancement present
  • MRI Brain:
  • Area postrema lesion (dorsal medulla)
  • No typical MS lesions (no periventricular Dawson's fingers)
  • MRI Orbits:
  • Bilateral optic nerve enhancement and edema, extending to optic chiasm
  • Serum:
  • Aquaporin-4 (AQP4) antibody (NMO-IgG): POSITIVE - diagnostic
  • MOG antibody: Negative
  • LP:
  • WBC: 85 (neutrophilic pleocytosis - more than MS)
  • Protein: 110 (elevated)
  • Oligoclonal bands: Absent (unlike MS)
  • Labs: ANA positive, anti-dsDNA positive (known SLE)

Diagnosis

Neuromyelitis Optica Spectrum Disorder (NMOSD) - AQP4-IgG seropositive

  • IPND 2015 criteria met:
  • Core clinical characteristic (optic neuritis + myelitis + area postrema syndrome)
  • AQP4-IgG positive

Management

Acute Attack Treatment:

  1. High-dose IV methylprednisolone 1 g daily for 5 days - initiated immediately
  2. Plasmapheresis (PLEX) - given severity and poor response to steroids after 3 days
  • 5-7 exchanges
  1. Consider IVIG if PLEX unavailable

Long-term Immunosuppressive Therapy:

  1. Essential - untreated NMOSD has high relapse rate and accumulating disability
  2. First-line approved therapies:
  • Eculizumab (complement C5 inhibitor)
  • Inebilizumab (anti-CD19)
  • Satralizumab (anti-IL-6 receptor)
  1. Off-label options:
  • Rituximab (anti-CD20)
  • Azathioprine
  • Mycophenolate mofetil
  1. Avoid MS drugs - interferon beta and fingolimod may worsen NMOSD

Symptomatic Management:

  1. Intractable hiccups: Baclofen, gabapentin, chlorpromazine
  2. Neurogenic bladder: Intermittent catheterization, anticholinergics
  3. Neuropathic pain: Gabapentin, pregabalin
  4. Spasticity: Baclofen, tizanidine

Rehabilitation:

  1. Intensive inpatient rehabilitation
  2. PT for mobility, OT for ADLs
  3. Low vision services for visual impairment

Prognosis:

  • More severe attacks and worse recovery than MS
  • 50% require walking aid within 5-10 years
  • Mortality higher than MS if untreated

Clinical Image

Image Description: Sagittal T2-weighted MRI of the cervical and thoracic spine showing longitudinally extensive transverse myelitis (LETM) spanning more than 3 vertebral segments, characteristic of neuromyelitis optica spectrum disorder.

Attribution: Image from Radiopaedia.org, Case courtesy of Dr. Yuranga Weerakkody. Licensed under CC BY-NC-SA 3.0. Source: https://radiopaedia.org/cases/neuromyelitis-optica-1


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