Internal Medicine · Year 3 · from Internal Medicine
Case 3: Sepsis Resuscitation and Reassessment
Patient Presentation
A 55-year-old man with no significant medical history presents with 2 days of fever, productive cough, and worsening shortness of breath. Chest X-ray shows right lower lobe consolidation. Initial vitals show: BP 95/60, HR 105, RR 24, T 38.9°C, SpO2 92% on room air. Lactate is 3.8 mmol/L. He receives 30 mL/kg crystalloid and ceftriaxone plus azithromycin.
Post-Resuscitation Assessment (2 hours later)
- Blood Pressure: 102/65 mmHg (MAP 77)
- Heart Rate: 95 bpm
- Respiratory Rate: 20/min
- Oxygen Saturation: 96% on 2L NC
- Urine output: 60 mL in past hour
- Repeat lactate: 2.4 mmol/L
- Mental status: Alert and oriented
Physical Examination Post-Fluids
- General: Improved, more alert
- Cardiovascular: Regular rhythm, warm extremities, capillary refill <2 seconds
- Pulmonary: Crackles at right base, improved work of breathing
- Extremities: No mottling
Clinical Image
Figure 3: Algorithm for sepsis resuscitation showing dynamic assessment of fluid responsiveness and perfusion markers.
Image Source: Educational illustration for teaching purposes.
Questions
- What markers indicate adequate resuscitation in this patient?
- A) Normal blood pressure only
- B) Improved MAP, decreasing lactate, adequate urine output, improved mental status, warm extremities
- C) Absence of fever
- D) Normal heart rate
- What is the role of dynamic measures of fluid responsiveness?
- A) Replacing clinical assessment
- B) Guiding further fluid administration to avoid both under- and over-resuscitation
- C) Determining antibiotic choice
- D) Predicting mortality
- What is the appropriate duration of antibiotic therapy for uncomplicated pneumonia with sepsis?
- A) 3 days
- B) 5-7 days
- C) 14 days
- D) 21 days
- What are the potential harms of excessive fluid resuscitation?
- When should corticosteroids be considered in septic shock?
Answers
- B) Improved MAP, decreasing lactate, adequate urine output, improved mental status, warm extremities - Multiple markers of perfusion should be assessed:
- MAP ≥65 mmHg
- Lactate clearing (this patient: 3.8 → 2.4 mmol/L)
- Urine output ≥0.5 mL/kg/hr
- Mental status improvement
- Normalization of skin perfusion (warm, not mottled)
- B) Guiding further fluid administration to avoid both under- and over-resuscitation - Dynamic measures include:
- Passive leg raise response
- Pulse pressure variation (mechanically ventilated)
- Stroke volume variation
- IVC collapsibility (limited accuracy)
- These help determine if patient will benefit from additional fluids vs. moving to vasopressors
- B) 5-7 days - For most pneumonia with sepsis, 5-7 days is sufficient if clinical improvement occurs. Procalcitonin can guide discontinuation. Longer courses may be needed for:
- Pseudomonas or MRSA
- Lung abscess or empyema
- Immunocompromised patients
- Harms of excessive fluid resuscitation:
- Pulmonary edema and worsening respiratory failure
- Peripheral edema and impaired wound healing
- Intra-abdominal hypertension/abdominal compartment syndrome
- Dilutional coagulopathy
- Increased mortality in some studies (FEAST trial, CLOVERS trial suggests equipoise)
- Need for prolonged mechanical ventilation
- Goal is "just enough" fluid to optimize perfusion
- Corticosteroid indications in septic shock:
- Consider if vasopressor requirements are escalating or persistently high
- Typically hydrocortisone 200 mg/day (50 mg IV q6h or 200 mg continuous infusion)
- Survival benefit in refractory shock (APROCCHSS trial)
- May speed shock reversal (ADRENAL trial)
- Do not routinely test cortisol levels
- Discontinue once vasopressors weaned
Learning Points
- Sepsis-3 definition requires infection plus organ dysfunction (SOFA score increase ≥2); septic shock adds hypotension requiring vasopressors and lactate >2 despite fluids.
- Hour-1 Bundle includes lactate measurement, blood cultures, antibiotics, and fluid resuscitation initiated within 1 hour.
- Norepinephrine is the first-line vasopressor for septic shock; target MAP ≥65 mmHg.
- Source control (e.g., drainage of abscess, removal of infected catheter, biliary decompression) is essential and should not be delayed.
- Reassessment of perfusion markers guides ongoing resuscitation; both under-resuscitation and over-resuscitation can cause harm.