Internal Medicine · Year 3 · from Internal Medicine

Case 1: Diabetic Ketoacidosis

Patient Presentation

A 28-year-old woman with type 1 diabetes mellitus presents with nausea, vomiting, and abdominal pain for 2 days. She reports increased thirst and urinary frequency over the past week. She admits to running out of her insulin 3 days ago and being unable to afford a refill. She denies fever, dysuria, or respiratory symptoms.

Vital Signs

  • Blood Pressure: 98/62 mmHg
  • Heart Rate: 118 bpm
  • Respiratory Rate: 28/min (Kussmaul respirations)
  • Oxygen Saturation: 99% on room air
  • Temperature: 37.2°C

Physical Examination

  • General: Ill-appearing, drowsy but arousable, fruity breath odor
  • HEENT: Dry mucous membranes, sunken eyes
  • Cardiovascular: Tachycardic, regular rhythm
  • Pulmonary: Deep, rapid respirations; lungs clear
  • Abdomen: Diffusely tender without rebound or guarding
  • Skin: Decreased turgor, no rashes
  • Neurologic: Alert and oriented, no focal deficits

Initial Workup

  • Glucose: 485 mg/dL
  • Sodium: 128 mEq/L
  • Potassium: 5.8 mEq/L
  • Chloride: 95 mEq/L
  • Bicarbonate: 8 mEq/L
  • BUN/Creatinine: 32/1.8 mg/dL
  • Anion gap: 25 mEq/L
  • Arterial pH: 7.18
  • Serum ketones: Large
  • Urinalysis: Glucose 4+, ketones 3+, no leukocytes

Clinical Image

Figure 1: Diagram illustrating Kussmaul respirations - deep, labored breathing pattern representing respiratory compensation for metabolic acidosis in diabetic ketoacidosis.

Image Source: Educational illustration for teaching purposes.

Questions

  1. What are the diagnostic criteria for diabetic ketoacidosis (DKA)?
  • A) Glucose >200, pH <7.35, bicarbonate <18, ketones positive
  • B) Glucose >250, pH <7.30, bicarbonate <18, ketones positive, anion gap >12
  • C) Glucose >400, pH <7.20, bicarbonate <15, ketones positive
  • D) Glucose >300, pH <7.25, bicarbonate <20, anion gap >10
  1. What is the corrected sodium in this patient?
  • A) 128 mEq/L
  • B) 132 mEq/L
  • C) 134 mEq/L
  • D) 136 mEq/L
  1. What is the most critical electrolyte to monitor during DKA treatment?
  • A) Sodium
  • B) Potassium
  • C) Calcium
  • D) Phosphorus
  1. Outline the initial management priorities in treating DKA.
  1. When should sodium bicarbonate be administered in DKA?

Answers

  1. B) Glucose >250, pH <7.30, bicarbonate <18, ketones positive, anion gap >12 - DKA is characterized by hyperglycemia (>250 mg/dL), metabolic acidosis (pH <7.30, bicarbonate <18), positive serum/urine ketones, and elevated anion gap (>12).
  1. C) 134 mEq/L - Corrected sodium = measured sodium + 1.6 × [(glucose - 100)/100]. Here: 128 + 1.6 × [(485-100)/100] = 128 + 6.16 ≈ 134 mEq/L. This corrects for dilutional hyponatremia from hyperglycemia.
  1. B) Potassium - Although serum potassium may be normal or elevated on presentation, total body potassium is depleted. Insulin administration drives potassium intracellularly, risking severe hypokalemia. Do not start insulin until K+ >3.3 mEq/L.
  1. Initial management priorities:
  • Fluids: Normal saline 1-1.5 L/hour initially; switch to 0.45% saline when sodium normalizes
  • Insulin: Regular insulin 0.1 units/kg bolus, then 0.1 units/kg/hour infusion (after confirming K+ >3.3)
  • Potassium: Add 20-40 mEq/L to fluids when K+ <5.3 mEq/L
  • Monitor: Glucose hourly, electrolytes every 2-4 hours
  • Dextrose: Add D5 to fluids when glucose <200-250 mg/dL
  • Search for precipitant: Infection, medication non-adherence, MI, etc.
  1. Bicarbonate administration:
  • Only indicated when pH <6.9
  • 100 mEq sodium bicarbonate in 400 mL sterile water with 20 mEq KCl over 2 hours
  • Routine bicarbonate administration is not recommended for pH ≥6.9
  • May cause paradoxical CNS acidosis and hypokalemia

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