Immunology · Year 2 · from Immunology

Case 1: Systemic Lupus Erythematosus

Patient Demographics

  • Age: 28 years old
  • Sex: Female
  • Ethnicity: African American

Chief Complaint

Joint pain, facial rash, and fatigue for 3 months

History of Present Illness

A 28-year-old woman presents with a 3-month history of progressive fatigue, joint pain, and a rash on her face. She describes morning stiffness lasting over an hour and symmetric pain in her hands, wrists, and knees without significant swelling. The facial rash appeared after sun exposure during a beach vacation and has persisted. She also reports hair loss, oral ulcers, and occasional low-grade fevers. Recently, she noticed swelling in her legs and foamy urine.

Past Medical History

  • Recurrent episodes of pleuritic chest pain over past year
  • History of photosensitivity
  • One miscarriage at 12 weeks gestation

Family History

  • Mother with rheumatoid arthritis
  • Maternal aunt with Sjogren's syndrome

Physical Examination

  • General: Fatigued-appearing woman in no acute distress
  • Vital Signs: T 37.8C, HR 92, RR 16, BP 148/94
  • Skin: Erythematous malar rash sparing nasolabial folds; diffuse alopecia; oral ulcers on hard palate
  • Musculoskeletal: Tenderness at MCPs, wrists, and knees bilaterally without synovitis
  • Cardiovascular: Normal S1/S2, no murmurs or rubs
  • Pulmonary: Decreased breath sounds at left base
  • Extremities: 2+ pitting edema to mid-calf bilaterally

Laboratory Workup

TestResultReference Range
WBC3,100/uL4,500-11,000/uL
Hemoglobin10.2 g/dL12-16 g/dL
Platelets98,000/uL150,000-400,000/uL
Creatinine1.4 mg/dL0.6-1.2 mg/dL
ANA1:640 homogeneousNegative
Anti-dsDNAPositive (high titer)Negative
Anti-SmithPositiveNegative
Complement C345 mg/dL (low)90-180 mg/dL
Complement C48 mg/dL (low)10-40 mg/dL
Urinalysis3+ protein, 2+ blood, RBC castsNormal
24-hour urine protein3.2 g<150 mg
Chest X-raySmall left pleural effusionNormal

Clinical Image

Image showing characteristic malar (butterfly) rash distribution pattern seen in systemic lupus erythematosus, typically sparing the nasolabial folds. Image source: Course lecture materials.

Diagnosis

Systemic Lupus Erythematosus (SLE) with Class IV Lupus Nephritis

Discussion

SLE is a prototypical systemic autoimmune disease characterized by autoantibodies against nuclear antigens and multi-organ involvement:

Pathogenesis:

  • Defective clearance of apoptotic cells exposes nuclear antigens
  • Loss of tolerance leads to autoantibody production (ANA, anti-dsDNA, anti-Smith)
  • Immune complex deposition causes inflammation via complement activation
  • Type I interferon signature amplifies the autoimmune response

This patient meets multiple classification criteria:

  • Malar rash (photosensitive)
  • Arthritis (non-erosive)
  • Serositis (pleuritis)
  • Renal involvement (nephritis with proteinuria and casts)
  • Hematologic (leukopenia, anemia, thrombocytopenia)
  • ANA positive
  • Anti-dsDNA and anti-Smith positive

Autoantibodies in SLE and their associations:

AutoantibodyAssociation
Anti-dsDNALupus nephritis, disease activity
Anti-SmithMost specific for SLE
Anti-Ro (SS-A)Neonatal lupus, photosensitivity
Anti-La (SS-B)Sjogren's overlap
AntiphospholipidThrombosis, pregnancy loss

Low complement levels indicate active immune complex disease.

Treatment

  1. Immediate: Renal biopsy to classify nephritis
  2. Induction therapy: Mycophenolate mofetil or cyclophosphamide plus corticosteroids for Class IV nephritis
  3. Maintenance: Mycophenolate mofetil with low-dose prednisone
  4. Hydroxychloroquine: All SLE patients (reduces flares, thrombosis, damage accrual)
  5. Sun protection: Strict photoprotection
  6. Monitoring: Anti-dsDNA titers and complement levels correlate with disease activity

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