Gastrointestinal · Year 2 · from Gastrointestinal

Case 1: Non-Variceal Upper GI Bleeding - Peptic Ulcer Disease

Patient Presentation

Demographics: 68-year-old male

Chief Complaint: Vomiting blood and black tarry stools for 1 day

History of Present Illness: The patient developed nausea and vomited approximately 500 mL of bright red blood mixed with clots this morning. He subsequently noticed black, tarry, foul-smelling stools. He reports epigastric discomfort for the past 2 weeks that he attributed to "indigestion." He felt lightheaded when standing and nearly fainted in the bathroom.

Past Medical History: Osteoarthritis (takes ibuprofen 800 mg three times daily for 3 months), hypertension, hyperlipidemia, coronary artery disease with prior stent (on aspirin 81 mg and clopidogrel)

Social History: Former smoker (quit 10 years ago), social alcohol (2-3 beers weekly)

Physical Examination

  • Vital Signs: BP 92/58 mmHg (supine), HR 112 bpm; Standing: BP 74/50 mmHg, HR 138 bpm (positive orthostatic hypotension)
  • General: Pale, diaphoretic male appearing anxious
  • Abdomen: Soft, mild epigastric tenderness, no peritoneal signs, hyperactive bowel sounds
  • Rectal Exam: Black, tarry, guaiac-positive stool

Workup and Results

  • CBC: Hemoglobin 7.2 g/dL (baseline 13.5), Platelets 185,000
  • BMP: BUN 48 mg/dL (elevated due to blood in GI tract), Creatinine 1.1 mg/dL
  • Coagulation: PT/INR normal
  • Type and Screen: Performed; 4 units PRBCs crossmatched
  • Glasgow-Blatchford Score: 14 (high-risk; hemoglobin, BUN, blood pressure, melena all contribute)
  • EGD (performed within 12 hours): Large 2-cm posterior duodenal ulcer with visible vessel (Forrest IIa) at the base; no active bleeding at time of endoscopy

Endoscopic image of a duodenal ulcer with evidence of recent hemorrhage. The visible vessel (Forrest IIa classification) at the ulcer base indicates high risk for rebleeding and requires endoscopic therapy.

Image Source: Wikimedia Commons. Samir, CC BY-SA 3.0

Diagnosis

Acute Upper GI Bleeding from Posterior Duodenal Ulcer (NSAID-induced) - Forrest IIa (High-Risk Stigmata)

Clinical Correlation

This case illustrates classic non-variceal upper GI bleeding from peptic ulcer disease, the most common cause of UGIB (30-40% of cases). Key teaching points include:

Risk Factor Analysis:

  • NSAID use: Direct mucosal injury and prostaglandin inhibition reduce mucosal defense
  • Dual antiplatelet therapy: Increases bleeding risk 3-5 fold when combined with NSAIDs
  • Location: Posterior duodenal ulcers are particularly dangerous due to proximity to the gastroduodenal artery, which can cause massive hemorrhage if eroded

Clinical Presentation:

  • Hematemesis: Frank blood indicates ongoing or recent active bleeding
  • Melena: Black tarry stool from degraded blood (indicates at least 50-100 mL blood loss)
  • Orthostatic hypotension: Indicates significant volume depletion (typically > 1L blood loss)
  • Elevated BUN:Creatinine ratio: Blood protein absorbed in GI tract increases urea production

Forrest Classification and Rebleeding Risk:

  • Forrest Ia (spurting): 55% rebleeding
  • Forrest Ib (oozing): 43% rebleeding
  • Forrest IIa (visible vessel): 43% rebleeding - requires therapy
  • Forrest IIb (adherent clot): 22% rebleeding - consider clot removal and therapy
  • Forrest IIc (flat pigmented spot): 10% rebleeding - no therapy needed
  • Forrest III (clean base): 5% rebleeding - no therapy needed

Treatment

Resuscitation:

  • Two large-bore IV access
  • Crystalloid resuscitation (Lactated Ringer's)
  • Restrictive transfusion strategy: Target hemoglobin 7-8 g/dL (not normal range)
  • ICU admission given hemodynamic instability

Pre-Endoscopic Management:

  • IV PPI: Pantoprazole 80 mg bolus followed by 8 mg/hour infusion
  • Hold antiplatelet agents temporarily (discuss with cardiology)
  • Consider erythromycin 250 mg IV to promote gastric emptying before EGD

Endoscopic Therapy (for Forrest IIa):

  • Combination therapy preferred: Epinephrine injection (1:10,000) followed by thermal coagulation (bipolar) OR endoscopic clips
  • Epinephrine alone is not sufficient (high rebleeding rates)

Post-Endoscopic Care:

  • Continue high-dose IV PPI infusion (80 mg bolus + 8 mg/hour) for 72 hours
  • Test for H. pylori (stool antigen or biopsy urease test)
  • Transition to oral PPI twice daily after 72 hours
  • NSAID cessation - if pain management needed, use acetaminophen or consider COX-2 selective agent with PPI if NSAID absolutely required
  • Cardiology consultation regarding timing of antiplatelet resumption (typically resume aspirin within 3-5 days; rebleeding risk vs. stent thrombosis)

All cases for this lecture as Markdown