Cardiovascular · Year 1 · from Cardiovascular

Case 3: Septic Shock with Distributive Physiology

Patient Presentation

Demographics: 72-year-old male

Chief Complaint: Fever, confusion, and low blood pressure

History of Present Illness: A 72-year-old male with history of diabetes and recent UTI (treated with oral antibiotics 5 days ago) presents with fever, confusion, and weakness for 1 day. His wife reports he has been "not himself" and has had difficulty urinating. He has had shaking chills and decreased oral intake.

Physical Examination:

  • Vital Signs: BP 75/40 mmHg (MAP 52), HR 125 bpm, RR 26/min, SpO2 92% on room air, Temp 39.2°C
  • General: Confused, flushed, appears ill
  • Cardiovascular: Tachycardic, warm extremities despite hypotension, bounding pulses
  • Lungs: Clear
  • Abdomen: Suprapubic tenderness
  • Extremities: Warm, well-perfused, no edema

Workup

  • Labs:
  • WBC: 22,000/μL with 15% bands (left shift)
  • Lactate: 5.2 mmol/L
  • Creatinine: 2.8 mg/dL (baseline 1.2)
  • Procalcitonin: 25 ng/mL (markedly elevated)
  • Urinalysis: Pyuria, bacteriuria
  • Blood cultures: Pending (later grew E. coli)
  • Invasive hemodynamics (after resuscitation):
  • Cardiac index: 4.8 L/min/m² (elevated)
  • SVR: 450 dynes·s/cm⁵ (severely reduced, normal 800-1200)
  • PCWP: 8 mmHg (low-normal)
  • CVP: 6 mmHg

Diagnosis

Septic shock secondary to urosepsis with distributive physiology

Cardiac Output Correlation: MAP = CO × SVR

In septic shock (distributive):

  • SVR severely reduced: Inflammatory mediators (NO, cytokines) cause profound vasodilation
  • Cardiac output increased: Compensatory increase to maintain perfusion (hyperdynamic state)
  • Despite high CO, hypotension: MAP = CO × SVR; if SVR drops dramatically, MAP falls despite high CO
  • Tissue hypoperfusion: Maldistribution of blood flow → elevated lactate despite high CO

Contrast with cardiogenic shock:

ParameterCardiogenicSeptic (early)
CO↓↓
SVR↑↑↓↓
PCWP↑↑Normal/↓
ExtremitiesColdWarm

Late septic shock: Myocardial depression may develop → CO falls → mixed picture

Treatment

  1. Aggressive fluid resuscitation: 30 mL/kg crystalloid within first 3 hours
  2. Broad-spectrum antibiotics: Ceftriaxone + metronidazole (started within 1 hour)
  3. Vasopressors: Norepinephrine (α1 agonist) to increase SVR and MAP
  4. Target MAP ≥65 mmHg
  5. Foley catheter (may need urology consult for obstruction)
  6. Source control: Remove any obstructed catheter, drainage if abscess
  7. Stress-dose steroids if vasopressor-refractory

Clinical Image

Image Description: Diagram illustrating the hemodynamic profile of septic shock showing decreased systemic vascular resistance (SVR) with compensatory increased cardiac output, resulting in warm shock physiology with hypotension.

Source: Wikimedia Commons - Septic shock physiology License: CC BY-SA 4.0 URL: https://commons.wikimedia.org/wiki/File:Septic_shock.svg

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